<?xml version="1.0"?>
<Articles JournalTitle="Acta Medica Iranica">
  <Article>
    <Journal>
      <PublisherName>Tehran University of Medical Sciences</PublisherName>
      <JournalTitle>Acta Medica Iranica</JournalTitle>
      <Issn>0044-6025</Issn>
      <Volume>33</Volume>
      <Issue>1-2</Issue>
      <PubDate PubStatus="epublish">
        <Year>1995</Year>
        <Month>06</Month>
        <Day>15</Day>
      </PubDate>
    </Journal>
    <title locale="en_US">AUTORADIOGRAPHIC STUDY OF CORNEAL NEOVASCULARIZATION INDUCED BY ALKALINE BURN</title>
    <FirstPage>12</FirstPage>
    <LastPage>17</LastPage>
    <AuthorList>
      <Author>
        <FirstName></FirstName>
        <LastName>Ali Ghadiri</LastName>
        <affiliation locale="en_US"></affiliation>
      </Author>
      <Author>
        <FirstName></FirstName>
        <LastName>Yin Shuguo</LastName>
        <affiliation locale="en_US"></affiliation>
      </Author>
    </AuthorList>
    <History>
      <PubDate PubStatus="received">
        <Year>2015</Year>
        <Month>09</Month>
        <Day>28</Day>
      </PubDate>
    </History>
    <abstract locale="en_US">Corneal neovasculariuulon (CNJ1 can cause significant visual loss. The kinetics of endothelial cells durin{: microvascular growth were studied usinga model of alkaline cauteriuuion-induced neovascularization ofthe 40 rabbits cornea. Changes in DNA synthetic activity of endothelial cells during neovasculanuuion were assessed by autoradiographic studyojJJl_TdR incorporation, In vivo and In vitro. Microscopic study demonstrated that migration and redistribution of existing endothelial cells from the limbal vessels enabled vascular sprouting and elongation with cellular proliferation.</abstract>
    <web_url>https://acta.tums.ac.ir/index.php/acta/article/view/1656</web_url>
    <pdf_url>https://acta.tums.ac.ir/index.php/acta/article/download/1656/1649</pdf_url>
  </Article>
</Articles>
